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Anterior, posterior, and nonkeratometric contributions to refractive astigmatism in pseudophakes.

PMID: 32815865 (view PubMed database entry)
DOI: 10.1097/j.jcrs.0000000000000390 (read at publisher's website )

Neal Kansara, David Cui, Kalins Banerjee, Zachary Landis, Ingrid U Scott, Seth M Pantanelli,

<h4>Purpose</h4>To investigate the relationship between measured anterior (ACA) and posterior (PCA) keratometric astigmatism and postoperative refractive astigmatism (RA) and to quantify noncorneal astigmatism (NCA) contributions to RA.<h4>Setting</h4>Penn State College of Medicine, Hershey, Pennsylvania, USA.<h4>Design</h4>Retrospective consecutive case series.<h4>Methods</h4>Consecutive eyes underwent preoperative biometry (IOLMaster 700) and tomography/topography using a dual Scheimpflug-placido disk-based device (Galilei G4), cataract surgery with implantation of a monofocal intraocular lens (IOL), and postoperative manifest refractions. RA was compared with keratometric astigmatism using the following methods: IOLMaster, SimK, CorT, SimK + measured PCA, total corneal power at the corneal plane (TCP2), and CorT(Total). An ocular residual astigmatism (ORA) vector was calculated between RA and each measured astigmatism.<h4>Results</h4>Analysis was based on 296 eyes. ORA centroids were 0.28 @ 179, 0.45 @ 001, 0.37 @ 001, 0.19 @ 003, 0.19 @ 001, and 0.23 @ 178 diopter (D) for the 6 aforementioned methods, respectively (P < .000001 [ORAx, ORAy]). Based on TCP2 measurements, eyes with against-the-rule ACA and with-the-rule (WTR) ACA had ORA centroids of 0.09 @ 082 and 0.58 @ 001 D (P < .000001 [ORAx, ORAy]), respectively. ORA was nonzero and not entirely explained by the cornea, especially in those with WTR ACA.<h4>Conclusions</h4>Total keratometric astigmatism did not explain all ocular astigmatism. Noncorneal contributions were significant, especially in eyes with WTR ACA.

J Cataract Refract Surg (Journal of cataract and refractive surgery)
[2021, 47(1):93-99]

Cited: 2 times

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